btg2 cdna Search Results


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Full length Clone DNA of Human BTG family, member 2 with N terminal Myc tag.
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DNAFORM Inc cdna of btg2 id: 40032675
Cdna Of Btg2 Id: 40032675, supplied by DNAFORM Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Genome Systems Inc cdna probes btg2 (genbank #h86711)
Cdna Probes Btg2 (Genbank #H86711), supplied by Genome Systems Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 90 stars, based on 1 article reviews
cdna probes btg2 (genbank #h86711) - by Bioz Stars, 2026-09
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Origen Biomedical full-length btg2 cdna
miR‐15a targets <t>BTG2</t> in ccRCC cells. (A) Targeting relation between miR‐15a and BTG2 verified by dual‐luciferase reporter gene assay (* p < 0.05; compared using two‐way ANOVA). (B) BTG2 expression in UMRC‐2 cells treated with exosomes determined by RT‐qPCR (* p < 0.05; compared using unpaired t test). (C) BTG2 expression in ccRCC tissues and adjacent normal tissues detected by RT‐qPCR (* p < 0.05; compared using paired t test). (D) Correlation analysis between miR‐15a and BTG2 in ccRCC tissues. (E) BTG2 expression in ccRCC cells and HK‐2 cells detected by RT‐qPCR (* p < 0.05; compared using one‐way ANOVA). (F) BTG2 expression quantified by RT‐qPCR following altered expression of miR‐15a (* p < 0.05, ** p < 0.01; compared using one‐way ANOVA). (G) Transfection efficiency of BTG2‐OE in cells verified by RT‐qPCR (* p < 0.05; compared using unpaired t test). (H) Effects of BTG2 on ccRCC cell proliferation determined by colony formation assay (* p < 0.05; compared using unpaired t test)
Full Length Btg2 Cdna, supplied by Origen Biomedical, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/btg2+cdna/full+length+btg2+cdna/pmc11896453-87-18-21
Average 90 stars, based on 1 article reviews
full-length btg2 cdna - by Bioz Stars, 2026-09
90/100 stars
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Full length Clone DNA of Mouse B cell translocation gene 2, anti-proliferative with N terminal Myc tag.
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Full length Clone DNA of Human BTG family, member 2 with C terminal HA tag.
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Full length Clone DNA of Mus musculus B cell translocation gene 2, anti-proliferative.
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Full length Clone DNA of Mouse B cell translocation gene 2, anti-proliferative with C terminal Myc tag.
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N/A
Full length Clone DNA of Mouse B cell translocation gene 2, anti-proliferative.
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N/A
Full length Clone DNA of Human BTG family, member 2 with C terminal Flag tag.
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Full length Clone DNA of Human BTG family, member 2 with C terminal Myc tag.
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Full length Clone DNA of Human BTG family, member 2 with C terminal GFPSpark tag.
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Image Search Results


miR‐15a targets BTG2 in ccRCC cells. (A) Targeting relation between miR‐15a and BTG2 verified by dual‐luciferase reporter gene assay (* p < 0.05; compared using two‐way ANOVA). (B) BTG2 expression in UMRC‐2 cells treated with exosomes determined by RT‐qPCR (* p < 0.05; compared using unpaired t test). (C) BTG2 expression in ccRCC tissues and adjacent normal tissues detected by RT‐qPCR (* p < 0.05; compared using paired t test). (D) Correlation analysis between miR‐15a and BTG2 in ccRCC tissues. (E) BTG2 expression in ccRCC cells and HK‐2 cells detected by RT‐qPCR (* p < 0.05; compared using one‐way ANOVA). (F) BTG2 expression quantified by RT‐qPCR following altered expression of miR‐15a (* p < 0.05, ** p < 0.01; compared using one‐way ANOVA). (G) Transfection efficiency of BTG2‐OE in cells verified by RT‐qPCR (* p < 0.05; compared using unpaired t test). (H) Effects of BTG2 on ccRCC cell proliferation determined by colony formation assay (* p < 0.05; compared using unpaired t test)

Journal: The Kaohsiung Journal of Medical Sciences

Article Title: Exosomal microRNA ‐15a from ACHN cells aggravates clear cell renal cell carcinoma via the BTG2 / PI3K / AKT axis

doi: 10.1002/kjm2.12428

Figure Lengend Snippet: miR‐15a targets BTG2 in ccRCC cells. (A) Targeting relation between miR‐15a and BTG2 verified by dual‐luciferase reporter gene assay (* p < 0.05; compared using two‐way ANOVA). (B) BTG2 expression in UMRC‐2 cells treated with exosomes determined by RT‐qPCR (* p < 0.05; compared using unpaired t test). (C) BTG2 expression in ccRCC tissues and adjacent normal tissues detected by RT‐qPCR (* p < 0.05; compared using paired t test). (D) Correlation analysis between miR‐15a and BTG2 in ccRCC tissues. (E) BTG2 expression in ccRCC cells and HK‐2 cells detected by RT‐qPCR (* p < 0.05; compared using one‐way ANOVA). (F) BTG2 expression quantified by RT‐qPCR following altered expression of miR‐15a (* p < 0.05, ** p < 0.01; compared using one‐way ANOVA). (G) Transfection efficiency of BTG2‐OE in cells verified by RT‐qPCR (* p < 0.05; compared using unpaired t test). (H) Effects of BTG2 on ccRCC cell proliferation determined by colony formation assay (* p < 0.05; compared using unpaired t test)

Article Snippet: The first five nucleotides complementary to the seed region of miR‐15a were mutated by site‐directed mutagenesis, and the full‐length BTG2 cDNA (OriGen Biomedical, Austin, TX) without the 3′‐UTR was subcloned into the eukaryotic expression vector PCMM‐ENTRY.

Techniques: Luciferase, Reporter Gene Assay, Expressing, Quantitative RT-PCR, Transfection, Colony Assay

BTG2 can offset the effects of miR‐15a on ccRCC cells. (A) Establishment of cells overexpressing both miR‐15a and BTG2 verified by RT‐qPCR (* p < 0.05; compared using unpaired t test). (B) Cell proliferation detected by colony formation assay (* p < 0.05; compared using unpaired t test). (C) Cell migration examined by transwell assay (* p < 0.05; compared using unpaired t test). (D) Cell invasion evaluated by transwell assay (* p < 0.05; compared using unpaired t test). (E) Cell apoptosis determined by flow cytometry (* p < 0.05; compared using unpaired t test). (F) EMT activity of cells shown by detecting the florescence intensity of E‐cadherin

Journal: The Kaohsiung Journal of Medical Sciences

Article Title: Exosomal microRNA ‐15a from ACHN cells aggravates clear cell renal cell carcinoma via the BTG2 / PI3K / AKT axis

doi: 10.1002/kjm2.12428

Figure Lengend Snippet: BTG2 can offset the effects of miR‐15a on ccRCC cells. (A) Establishment of cells overexpressing both miR‐15a and BTG2 verified by RT‐qPCR (* p < 0.05; compared using unpaired t test). (B) Cell proliferation detected by colony formation assay (* p < 0.05; compared using unpaired t test). (C) Cell migration examined by transwell assay (* p < 0.05; compared using unpaired t test). (D) Cell invasion evaluated by transwell assay (* p < 0.05; compared using unpaired t test). (E) Cell apoptosis determined by flow cytometry (* p < 0.05; compared using unpaired t test). (F) EMT activity of cells shown by detecting the florescence intensity of E‐cadherin

Article Snippet: The first five nucleotides complementary to the seed region of miR‐15a were mutated by site‐directed mutagenesis, and the full‐length BTG2 cDNA (OriGen Biomedical, Austin, TX) without the 3′‐UTR was subcloned into the eukaryotic expression vector PCMM‐ENTRY.

Techniques: Quantitative RT-PCR, Colony Assay, Migration, Transwell Assay, Flow Cytometry, Activity Assay

miR‐15a‐mediated BTG2 inhibition increases activity of the PI3K/AKT pathway. Changes of PI3K/AKT pathway in exosomes‐treated cells or cells overexpressing BTG2 or/and miR‐15a detected by western blot analysis

Journal: The Kaohsiung Journal of Medical Sciences

Article Title: Exosomal microRNA ‐15a from ACHN cells aggravates clear cell renal cell carcinoma via the BTG2 / PI3K / AKT axis

doi: 10.1002/kjm2.12428

Figure Lengend Snippet: miR‐15a‐mediated BTG2 inhibition increases activity of the PI3K/AKT pathway. Changes of PI3K/AKT pathway in exosomes‐treated cells or cells overexpressing BTG2 or/and miR‐15a detected by western blot analysis

Article Snippet: The first five nucleotides complementary to the seed region of miR‐15a were mutated by site‐directed mutagenesis, and the full‐length BTG2 cDNA (OriGen Biomedical, Austin, TX) without the 3′‐UTR was subcloned into the eukaryotic expression vector PCMM‐ENTRY.

Techniques: Inhibition, Activity Assay, Western Blot